2018
Hunger is not a hindrance for N-terminal acetylation
Arnesen Lab reveals that the global level of N-terminal acetylation, a highly abundant protein modification in eukaryotes, remains stable in starving yeast cells despite an overall decrease in the cellular level of acetyl-CoA.
Photo: Leo Rivas Copyright: Unsplash
A mutation that can cause autism
An international collaboration effort have identified several new mutations in NAA15, a known risk gene for Autism Spectrum Disorder. This work gives new insight into the heredity of ASD and may lead to better treatment of patients with neurodegenerative disorders.
Putting a break on cell movement
Adrian Drazic, Henriette Aksnes and Michaël Marie from Thomas Arnesen's group found an enzyme that others have been looking for for 30 years.
The FEBS Congress 2018
The NAT group was well represented at this years FEBS Congress.
ERC Consolidator Grant
Arnesen received an ERC Consolidator Grant for the period 2018 to 2023.
UIB.no: ERC consolidator grant to Thomas Arnesen
Forskningsrådet.no: Banebrytende forskning ble belønnet [Norwegian]
Påhøyden.no: Får høythengende EU-midler [Norwegian]
2017
Hangin’ out at the Golgi
Naa60 and the means by which it associates with the Golgi membrane caught the interest of researchers in the Arnesen group UiB who recently presented their findings in the Journal of Biological Chemistry.